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Keao Xieli Co Ltd normal chow diet (ncd)
Normal Chow Diet (Ncd), supplied by Keao Xieli Co Ltd, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/normal+chow+diet+(ncd)/normal+chow+diet+ncd/pmc12113151-26-2-9
Average 90 stars, based on 1 article reviews
normal chow diet (ncd) - by Bioz Stars, 2026-11
90/100 stars

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Related Articles

other:

Article Title: Hepatic but not Intestinal FBP1 Is Required for Fructose Metabolism and Tolerance.
Article Snippet: The mice were fed ad libitum with normal chow diet (NCD, Beijing Keao Xieli Feed Co., Ltd., Beijing, China) or HFrD (D11707R, 65% fructose diet, Research Diets, USA).

Article Title: Carbon Dots as Potential Therapeutic Agents for Treating Non-Alcoholic Fatty Liver Disease and Associated Inflammatory Bone Loss.
Article Snippet: C57BL/6J mice were fed with normal chow diet (NCD, n = 5) which supplies 13% calories from fat, 24% calories from protein and 63% calories from carbohydrates (Keao Xieli Feed Co., Ltd., Beijing, China), or high-fat diet (HFD, n = 20) which 4 supplies 60% calories from fat, 20% calories from protein and 20% calories from carbohydrates (Dyets Inc., Bethlehem, PA USA).

Control:

Article Title: Zonated iron deposition in the periportal zone of the liver is associated with selectively enhanced lipid synthesis.
Article Snippet: 1State Key Laboratory of Environmental Chemistry and Ecotoxicology, Research Center for Eco-Environmental Sciences, Chinese Academy of Sciences, Beijing, China 2University of Chinese Academy of Sciences, Beijing, China 3Liver Research Center, Beijing Friendship Hospital, Capital Medical University, Beijing, China 4Beijing Key Laboratory of Translational Medicine in Liver Cirrhosis and National Clinical Research Center of Digestive Diseases, Beijing, China 5The First Affiliated Hospital of Shandong First Medical University, Jinan, China 6Ministry of Education Key Laboratory of Molecular and Cellular Biology, The Key Laboratory of Animal Physiology, Biochemistry and Molecular Biology of Hebei Province, College of Life Sciences, Hebei Normal University, Shijiazhuang, Hebei Province, China 7The Fourth Affiliated Hospital, The First Affiliated Hospital, Institute of Translational Medicine, School of Public Health, Cancer Center, State Key Laboratory of Experimental Hematology, Zhejiang University School of Medicine, Hangzhou, China

Fat:

Article Title: Glucose-induced CRL4 COP1 -p53 axis amplifies glycometabolism to drive tumorigenesis.
Article Snippet: In brief Su et al. found that glycometabolism selfamplifies via a glucose-induced posttranslational modification cascade culminating in CRL4-mediated degradation of the tumor suppressor p53, thus reinforcing the Warburg effect and overcoming tumor suppression.. Such mutation-independent p53 checkpoint bypass may represent the carcinogenic origin and targetable vulnerability of hyperglycemia-driven cancer.



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